If the heart survives after an ischemic heart attack, there are ways that it copes. The structural scaffold fibroblast cells remain, after the functional cells have died. These work much harder to maintain the akinetic region which is better than overt rupture, which would be lethal. The rest of the remaining heart undergoes stress and strain to maintain the difference in work, but at a cost.
Distinctive ERK and p38 signaling in remote and infarcted myocardium during post-MI remodeling in the mouse.
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